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Chronic polyradiculoneuritis, or chronic inflammatory demyelinating polyradiculoneuritis, (CIDP) is an autoimmune disease affecting the peripheral nervous system. This disease is classified under the more general name of peripheral neuropathy. IPDC is a close relative of Guillain-Barré syndrome (or Acute Idiopathic Polyradiculoneuropathy) and is considered its chronic counterpart. Its symptoms are also similar to progressive inflammatory neuropathy, a highly asymmetric variant of CIDP, known as Lewis-Sumner syndrome. The prevalence, estimated between 1/200,000 (children) and 1-7 / 100,000 (adults), is undoubtedly underestimated.
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The diagnosis is based on a set of criteria from: . The treatment depends on the severity of the CIDP, the age, the general condition of the patient and the contraindications to the following validated treatments: . Patients with purely motor CIDP should be given intravenous immunoglobulin infusions rather than steroids. In more moderate forms, clinical observation via an electroneuromyogram (ENMG), or even corticosteroid therapy are indicated. The combination of steroids and immunoglobulin infusion are possible.
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The reason for relapses and remissions varies considerably from patient to patient. A period of relapse can be very disturbing, but most patients make a significant recovery. The prognosis is generally good. If it is early, the initiation of treatment to prevent loss of nerve axons is recommended. However, many people end up with residual numbness, weakness, tremors, fatigue and other symptoms that can lead to a reduced quality of life.
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The sheath consists of multiple layers that grow back gradually; the myelin has to have a particular thickness prior to the nerve cells recapturing its ability to transmit impulses.There is no feasible way in predicting which nerves will regenerate.Research on the use of treatments that speed up the growth of motor nerves is under way but no one will know when they will arrive or if they will ever arrive.The area could function again, and it may seem as if the muscle has regained full strength, but the muscle and nerves have to work harder to carry out the same job and they end up tiring faster than was the case prior to GBS.These facets decrease the nerve signal transmission speed forever, after the patient has recovered from GBS.
Lastly, the treatment of GBS entails providing supportive care for patients with paralyzed diaphragms and intravenous injections of immunoglobulin for stable patients.In addition, administration of plasmapheresis is recommended.This essay presents the etiology, pathogenesis, diagnosis, clinical manifestations, prognosis, and the treatment of Rheumatoid Arthritis (RA) and Guillain-Barre Syndrome (GBS).However, most inflammatory conditions are treatable and preventable.The essay presents a detailed discussion on two inflammatory conditions, which are caused by immune responses that target cells, tissues, and organs in the body.
Incurable, symptomatic management and treatment is vital for patients with MS in order to provide a sustained quality of life.Binding to integrins prevents the association of adhesion molecules on endothelial cell surface, blocking migration of lymphocytes into the CNS, resulting in reduced numbers of T-cells in the CSF (Kargiotis, Paschali, Messinis & Papathanaspolous, 2010).Meletis and Zabriskie (2007) suggest that in relation to pharmacotherapy, the use of anti-oxidants, vitamins and change in diet directly affect cyokine production and the inflammatory response as well as the affecting the immune system.Multiple sclerosis [MS] is a chronic, autoimmune, inflammatory, neurodegenerative condition of the central nervous system [CNS] ...
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